Invest antiglaucoma drugs on apoptotic marker expression by human trabecular cells. In caused conjunctival leukoplakia in one case (21). When the mean apoptosis rates of the second and the third grwere significantly higher (p<0.05) (Fig.
All slides were examined by two masked observers. It is designated chemically as 9-fluoro-11β,17,21-trihydroxy-16α-methylpregna-1,4-diene-3,20-dione. 0000044799 00000 n
Conventional histological sections, pretreated with protease, were nick end labeled with biotinylated poly dU, introduced by terminal deoxy-transferase, and then stained using avidin-conjugated peroxidase. The plaque surfaces had numerous smooth tentacle-like projections and matching pits in the underlying epithelium.
Withdrawal of the drug and application of all-trans retinoic acid ointment resulted in resolving of the keratinisation.
and infiltrative lymphocytes at different stages of intestinal epithelia malignant transformations and further to analyze On the other hand, Brasnu The results of our study support these findings.
Light and dark cells were present Objective To explore the molecular mechanism of Fas counterattack by observing the expression of Fas and FasL and apoptosis of the epithelium Clinical tests and impression cytology showed ocular surface damage in patients using preserved antiglaucoma medications.
Cell sizes ranged from 3 to 20 micron across. Graefes Arch Clin Exp Ophthalmol HLA-DR in glaucoma patients and its influence on the outmorphologic effects of antiglaucoma drugs on the conjuncdouin C. Quaternary ammoniums and other preservatives’ contribution in oxidative stress and apoptosis on Chang conjunctival cells. These plaques were only found in FEC lenses and corresponded both in size and location to the structure known as the deep or amorphous layer of the lens capsule. 0000016258 00000 n
The investigation revealed round discoid plaques closely adherent to the capsule. This classification is based on the nucleus-to-cytoplasm ratios of epithelial cells and the numbers of goblet cells, with grade 2 considered abnormal. In this cross-sectional study, 109 participants (79 patients with topical medication and 30 untreated controls) completed the Ocular Surface Diseases Index (OSDI) questionnaire and underwent an ophthalmic examination, including Schirmer test, tear film breakup time (TBUT), and fluorescein staining. It is possible this may enhance the risk of external bleb scarring and filtration surgery failure. 0000076401 00000 n
30 tubulovillous adenomas, and 24 tubular adenomas. The OSDI did not discriminate between the two groups. ration of medication and mean apoptosis rate. 0000001901 00000 n
The mean PEG = pseudoexfoliative glaucoma; POAG = primary open-angle glaucoma.apoptosis rate of patients receiving medications for a period of 12 months or less was 0.589 ± 0.362 (0.052-0.960), 0.408 ± 0.135 (0.026-0.962) for patients receiving medication for 13-36 months, and 0.601 ± 0.343 (0.053-0.929) cantly higher when compared to the rates obtained in the While the mean apoptosis rate was 0.482 ± 0.392 (0.109-0.392) in patients with primary open-angle glaucoma (POAG), it was 0.580 ± 0.357 (0.356-0.986) in pseudoexfoliative glau2 groups was not statistically significant (p>0.05). 0000002535 00000 n
In the prtions on conjunctival epithelial cell expression of inflammatory cytokines such as interleukins 6, 8, and 10 used impression cytology and flow cytometry and showed that The effects of the duration of the medication on conjunctival epithelium were also studied with the number of medifects of long-term medications on conjunctival cell profile of medication longer than 3 years caused significant irreversible subclinical changes.
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