Will do more reading also.I’m having the same problem. Or possibly even stop the finasteride whilst cycling sarms?1) There are plenty of potential downsides to SARMs. It came to my attention that there was a thread made referring to my first video addressing Testosterone’s inherent ability to cause hair loss, and how they believe that Dutasteride in conjunction with TRT is the way to maintain “DHT fulfilled” physiological functions, as well as completely prevent hair loss. Topical Dutasteride, 1%, amazing results
Will have to look into MK. I had no erectile dysfunction whatsoever.I didn't lose any muscle at all while using Dutasteride.Minimal conversion of Testosterone to DHT occurs in muscle tissue, and the entirety of the human body’s anabolic effects in muscle tissue are mediated by endogenous Testosterone, not DHT.My priorities have changed over the years as I stopped caring about bodybuilding as much and started caring more about my hair.I started to give 5-alpha reductase inhibitors more of a chance in my research and experiments.I used to think they were the devil but I started to research more and realize what the androgenic component of certain anabolics really accomplishes in the body and how it really plays out in the whole cascade of events leading to eventual hair loss progression.I would have kept using Dutasteride if it worked the way I predicted it was going to work in 2017 before the experiment, but it didn't inhibit my systemic Testosterone at all from inducing androgenic activity at the hair follicle.I self-administered a dosage that pushed my Testosterone levels up to about 2000 ng/dL on purpose.I already know I’m severely prone to hair loss at that dosage with no protection, and I had to test the true extent of Testosterone’s own androgenic activity with DHT totally taken care of to know for sure what was going on in the body and to confirm my suspicions.It never did, and Testosterone’s lack of anabolic selectivity in the body was revealed and quite obvious once you take into account that my facial hair, body hair and hair loss was nearly unaltered in contrast to what a slightly lower level of Testosterone in my body with DHT present would induce.While Dutasteride and exogenous Testosterone therapy may be sufficient for many men in the short-term, ultimately hair loss will still be occurring, just at a less expedited rate than if DHT was present, and for those who are very sensitive to Testosterone, it may make things worse (e.g. Ok, my father is bald and I need to use finasteride right now but my hair loss is completely controlled now..Derek, are you still taking Dutesteride or did you go back to just the RUHi Derek be interested on your thoughts on taking sarms whilst still on finasteride.Ok, with the somewhat DHT blocking effect you mentioned on a recent post on Sarms/hairloss warning and with the potential muscle gains there seems to be not a lot of downside to MK 2866 or similar. Anti androgens can increase chance of heart failure.
The first thing I want to make clear in regards to my I don’t want anyone to misinterpret my videos and articles exhibiting the It is several-fold more androgenic than Testosterone.I think I made that pretty clear in my content, but just to clarify before we delve into this.The whole point of my article and video addressing Testosterone’s androgenicity was to state that endogenous androgens as a whole need to be addressed, NOT just the DHT.It came to my attention that there was a thread made referring to my first video addressing Testosterone’s inherent ability to cause hair loss, and how they believe that Dutasteride in conjunction with TRT is the way to maintain “DHT fulfilled” physiological functions, as well as completely prevent hair loss.The person who started that thread was essentially proposing what I believed in 2017.Back in 2017 I thought to myself okay; if you use Dutasteride you're guaranteed to be protected from hair loss from Testosterone because the 5-alpha reduction to DHT is taken care of.I realized some very crucial things shortly thereafter.While using Finasteride, you can reduce systemic DHT by roughly 70%, but you will still have 30% of your systemic DHT left in tact, which is still going to wreak havoc on your hair (common knowledge).This also is irrelevant to Finasteride’s 41% reduction of scalp DHT in particular, but that’s an entire topic in itself [Finasteride is a bandaid at most, but most guys know that.All you’re doing with Finasteride is putting your ticking time bomb of hair loss in slow motion.For many guys this is more than sufficient to stave off horrible hair loss for another several decades, but the fact remains that androgenic alopecia still occurs, albeit much slower while on Finasteride.My blood work indicated near 100% suppression of my systemic DHT while on 1 mg of Dutasteride per day.The blood work couldn't even read my DHT because it was below the detection limit.Even with a supraphysiological amount of Testosterone in my system from administering an exogenous a source of it and pushing my Testosterone up to 2,000 ng/dL (on purpose) I still had nearly zero DHT in my system because Dutasteride is just that effective at inhibiting 5-alpha reductase.After determining that 5-alpha reductase inhibition is the same regardless of how much Testosterone is present, I hypothesized that if I just crushed my DHT to zero it would be the equivalent of castration, and then I could just fulfill the physiological anabolic properties that I would normally get from androgens via Testosterone administration.This opened up the possibility to simply increasing exogenous Testosterone usage to whatever dosage would be necessary to fulfill whatever potential androgenic disparity there would be in the body with a complete lack of DHT.Based on my blood work, Dutasteride still inhibited Testosterone conversion enough to the point where I could hypothetically use unlimited amounts of Testosterone and still inhibit 5-alpha reduction into DHT completely.I thought okay great, so I can just use more Testosterone and keep all of my hair because DHT is still absent regardless of how much my Testosterone goes up.Even once I accomplished DHT inhibition, I had continuous androgenic side effects just from testosterone.The entirety of the experiment I had zero DHT, but I still experienced facial hair growth and had to shave my face just as often.I still experienced body hair growth, and despite it somewhat notably being reduced on my lower back area, I still had to manscape once a week to keep my body hair growth under control in general.These are things I had to do anyways just as frequently before when I had normal amounts of DHT in my body.I still had significant androgenic activity in my body, even with 0 DHT present, because Dutasteride does absolutely nothing to protect hair follicles from Testosterone, and actually increases scalp concentrations of Testosterone, which is why a minority of men who are especially sensitive to Testosterone actually experience exacerbated hair loss and what is often perceived to be chronic telogen effluvium while on Dutasteride.With this being said, Dutasteride will massively slow down hair loss in the majority of individuals, but it still doesn’t address the true root of the issue, and is not a solution for many.The first thing that I think needs to be taken into consideration that's largely overlooked is the fact that Finasteride and Dutasteride were created to address prostate size.The intention of the drug was not to address hair loss, the creators simply realized that a fringe benefit of inhibiting prostate growth by systemically inhibiting DHT conversion is that your hair can grow back because there's less androgen induced miniaturization occurring.If you reference the clinical data on Testosterone itself, you can clearly see that it stimulates prostate growth itself inherently, just to a lesser extent than DHT [Not relating to DHT conversion whatsoever, but on its own entirely.This is typically assessed in preclinical animal models when analyzing the efficacy of possible hormone replacement therapy alternatives by comparing a drugs stimulation of muscle growth relative to the stimulation of prostate and seminal vesicles growth.The less favorable the myotrophic-to-androgenic ratio is of a drug, the less ideal of a hormone replacement therapy option it is for men seeking to minimize androgenic alopecia.In general, the more androgenic a hormone is, the more it will increase prostate size and cause hair loss.On the opposite side of the spectrum, the less androgenic a hormone is, the less it will stimulate prostate growth, and the less it will expedite androgenic alopecia.Lowering androgenic activity in the body is totally counterproductive to being a male, and presents the paradox men face with their endocrine system.If you're a male, just the fact that you're a male puts you in a position where your hormone profile is counterproductive to keeping hair on your head, and the hormone profile necessary for complete hair loss prevention is counterproductive to being a male.What I mean by that is you rely on endogenous anabolic androgenic steroids for libido, penile sensitivity, muscle growth, recovery, neuroprotective effects on the brain, maintenance of masculine traits, and many more functions or traits that are fulfilled directly by androgens or androgen receptor activation.What most guys don’t realize is that Testosterone itself is the sole reason you can even operate and function without any DHT in your system.The reason is literally because Testosterone is androgenic itself.At best, it has that two-to-one selectivity for muscle tissue to prostate, and many other studies indicate that a two-to-one ratio is extremely generous.That's great because it fulfills the androgenic properties that would otherwise be fulfilled by DHT.Hence why most guys can use Dutasteride with no side effects, and those that experience androgen deficient side effects simply have free-testosterone levels that are too low to fulfill that androgen deficit, or do not understand effective Estrogen/general hormone management.The side effects would predictably be mitigated with exogenous androgen administration, or Estrogen management in almost all cases, dependent on what the root of the issue was specifically.But, the problem with that is that by fulfilling those very androgen dependent physiological functions; you will also miniaturize hair follicles.Maximizing libido, maximizing the sensitivity of the penis and maintaining masculine characteristics all must be achieved via sufficient androgen levels, which will also cause the miniaturization of hair follicles, and is exactly why Finasteride works so well.Finasteride directly reduces prostate size by inhibiting DHT, which the majority of the population has acknowledged is also what prevents progression of hair loss.If you look at androgen activity from the opposite side of the spectrum you will note that increased androgen levels in the body result in increased libido, increased penile sensitivity, increased facial hair growth, increased prostate size, and predictably, increased hair loss miniaturization.It doesn’t matter what androgenic hormone is in the body, whether its pure DHT, Drostanolone, Trenbolone, Anadrol, Dianabol, etc.
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